Our Specialists for Chronic Subdural Hematoma

Chronic subdural hematomas are the bread and butter of emergency neurosurgery, but the right operation for the right patient is not always obvious — especially in elderly patients on blood thinners or those whose hematomas keep coming back. Our trauma and neurovascular teams work together daily, offering the full range of options from bedside twist-drill drainage to middle meningeal artery embolization.

Dr. Das
Paramita Das, M.D., M.S.
Director, Neurotrauma; Director, Neurosurgical Trauma Fellowship

Dr. Das is Director of Neurotrauma at UChicago's Level 1 Trauma Center and directs the Neurosurgical Trauma Fellowship, one of only a few dedicated neurosurgical trauma fellowships in the country. She manages both head trauma and acute spine pathology and has been named to the Bucksbaum-Siegler Institute for Clinical Excellence. She trained at the University of Minnesota for residency and completed a skull base fellowship at Cleveland Clinic. As Director of Surgical Neurotrauma at UChicago, Dr. Das sees more chronic subdural hematomas than any other surgeon on the team, and she leads the decision-making on which patients get a bedside twist-drill versus a formal burr hole versus a combined procedure with MMA embolization. Her research focuses on outcomes and biomarkers after traumatic brain injury in older adults, the exact population most affected by cSDH.

JZ
Jehad Zakaria, M.D.
Endovascular Neurosurgeon

Dr. Zakaria is an endovascular neurosurgeon performing catheter-based treatment of stroke, brain aneurysms, and vascular malformations. He practices alongside the department's expanding cerebrovascular program, contributing to the regional thrombectomy and neurovascular service at UChicago. Dr. Zakaria is one of the endovascular neurosurgeons who performs middle meningeal artery embolization at UChicago, and he has published on technical refinements of the procedure using modern large-bore catheters (Interventional Neuroradiology, 2024). For patients whose chronic subdurals keep coming back, he is often the surgeon who closes off the blood supply once and for all.

Dr. Raksin
P. B. Raksin, M.D.
Associate Program Director, Neurological Surgery Residency

Dr. Raksin is an Associate Professor of Neurological Surgery and serves as Associate Program Director of the UChicago Neurological Surgery residency program. She is a senior clinician within the department and contributes broadly to resident education and general neurosurgical care. Dr. Raksin is one of the country's most respected voices in acute care neurosurgery — she co-edited the Atlas of Emergency Neurosurgery and Acute Care Neurosurgery by Case Management, both of which include detailed chapters on chronic subdural management. If you have a chronic subdural hematoma at UChicago Medicine or our partner trauma centers, she is one of the attendings most likely to be at the bedside.

What Is a Chronic Subdural Hematoma?

A chronic subdural hematoma is a collection of old blood that sits on the surface of the brain, underneath the tough outer covering called the dura. Unlike the acute bleeds you see after a bad car accident, a chronic subdural builds up slowly — typically over two to six weeks — and the blood inside has usually broken down into a thin, motor-oil-colored fluid by the time it is discovered.

These bleeds usually start when the small bridging veins that run between the brain and the dura get stretched and tear. In older adults, the brain naturally shrinks a little with age, which pulls those veins tight and makes them much more vulnerable. A trip on a rug, a bumped head on a cabinet, or even no clear trauma at all can be enough to start one — and patients often do not remember the event.

Chronic subdural hematoma has become one of the most common reasons older adults end up in a neurosurgeon's office. As the population ages and more people take blood thinners for heart and stroke prevention, the number of cases keeps climbing. Some experts now call it the most common neurosurgical condition of the elderly.

The encouraging news is that, once recognized, cSDH is usually very treatable. Most patients walk out of the hospital within a few days of surgery, and the majority return to their previous level of function.

At a Glance

  • A chronic subdural hematoma (cSDH) is old, liquefied blood that collects between the brain and its outer covering, the dura
  • It is most common in adults over 65, often after a fall or minor head bump weeks earlier — and even more common in patients on blood thinners
  • Symptoms can be subtle: headaches, confusion, unsteady walking, or weakness on one side that mimics a stroke
  • The workhorse treatment is burr hole drainage with a subdural drain, which resolves most cases in a single 30-minute procedure
  • Middle meningeal artery (MMA) embolization is a new minimally invasive add-on that roughly halves the chance the hematoma will come back
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What Does It Feel Like?

Chronic subdural hematomas are sneaky. Because the bleed builds up slowly, the brain has time to accommodate the pressure, and early symptoms can look like almost anything — dementia, a small stroke, a medication side effect, or just "slowing down with age." That's why family members are often the first to notice that something is off.

Common symptoms

When to go to the ER

Any older adult — especially someone on a blood thinner — who develops new headache, new weakness, new confusion, or a change in walking after a fall (even weeks earlier) should get a head CT. It takes 10 minutes and it can be life-saving.

How Is It Diagnosed?

The diagnosis is almost always made with a non-contrast CT scan of the head, which is fast, widely available, and does not require a needle. On CT, a chronic subdural typically appears as a crescent-shaped collection draped over the surface of the brain. The color of the blood on the scan tells the story of how old it is: bright white means fresh, dark gray means old and liquefied, and mixed densities mean the hematoma has been bleeding again on top of an older collection.

An MRI is sometimes added to sort out tricky cases — for example, when the hematoma is small, when there are membranes dividing the collection into compartments, or when the surgeon is trying to decide whether it will drain easily through a small opening.

Along with imaging, the team will:

Small, asymptomatic chronic subdurals in patients with no blood thinners can sometimes be managed without surgery, with a repeat scan in a few weeks. Symptomatic ones almost always need to be drained.

How Is It Treated?

Treatment has two goals: get the old blood out, and keep it from coming back. For decades the first goal was the focus of attention. In the past few years, a new set of randomized trials has transformed how we think about the second.

Burr hole drainage — the workhorse

For most symptomatic patients, the first-line treatment is burr hole craniostomy with placement of a subdural drain. Under anesthesia, the surgeon makes one or two small openings in the skull — each about the size of a nickel — washes out the old blood, and leaves a soft drain in place for one to two days. It is a short procedure (often under an hour), the recovery is usually fast, and most patients go home within three to five days.

Two landmark trials refined this operation. The Cambridge drain trial (Santarius, Lancet 2009) showed that leaving a drain for 48 hours cut the recurrence rate nearly in half and reduced mortality at 6 months. More recently, the FINISH trial (Raj, Lancet 2024) showed that irrigating the subdural space with saline during the operation also reduces reoperation rates compared to drainage alone. Both steps — irrigation and a drain — are now standard at UChicago.

Twist drill craniostomy and SEPS

For frail patients, those who cannot tolerate general anesthesia, or those whose hematomas are thin and free-flowing, there are even smaller procedures:

These techniques have lower up-front risk than a burr hole, but they also have somewhat higher recurrence rates — which is exactly where the next technique comes in.

Middle meningeal artery (MMA) embolization — the biggest advance in a generation

For as long as neurosurgeons have been draining subdurals, about 10 to 20 percent of patients have needed a second operation because the hematoma came back. The culprit is the outer membrane of the hematoma, which grows a web of fragile new blood vessels that keep re-bleeding. Those vessels are fed by the middle meningeal artery — an artery that runs on the inside of the skull and can be reached through a catheter from the groin or wrist.

In MMA embolization, an endovascular neurosurgeon threads a tiny catheter up to the middle meningeal artery and injects liquid embolic material or small particles to close off those feeder vessels. The procedure takes about an hour, involves no incision in the head, and most patients go home the next day.

In the past year, three major randomized trials have established MMA embolization as a true game-changer:

At UChicago, we now routinely offer MMA embolization — either as an adjunct to surgery for patients with a high risk of recurrence (big hematomas, bilateral hematomas, patients on lifelong blood thinners) or, occasionally, as a stand-alone treatment for smaller, less symptomatic collections.

What about dexamethasone or other medications?

Steroids were tried for years on the theory that inflammation drives hematoma regrowth. The Dex-CSDH trial (Hutchinson, NEJM 2020) settled the question: patients who received a two-week dexamethasone taper had worse functional outcomes and more complications than those who got placebo, even though they had slightly fewer reoperations. Routine steroid use for chronic subdurals is no longer recommended.

Managing blood thinners

One of the most delicate parts of treating chronic subdural hematoma is deciding what to do about the blood thinners that contributed to the bleed in the first place. We work closely with cardiology, stroke, and hematology to individualize this — pausing and reversing medications around surgery, then carefully restarting them at the right time to minimize the risk of both re-bleeding and stroke.

Second Opinion

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What Are the Outcomes?

Most patients with chronic subdural hematoma do well. With prompt surgery, symptoms usually improve within days to weeks, and the majority of patients return to their previous level of independence. That said, cSDH is not a benign disease — it is a marker of aging, frailty, and fall risk, and the long-term survival numbers reflect that as much as they reflect the surgery itself.

Reoperation rates

The rate at which a chronic subdural hematoma comes back and requires a second drainage has been the central problem in the field for decades. Here is what the modern data show:

TreatmentReoperation rateSource
Burr hole, no drain~24%Santarius, Lancet 2009
Burr hole with drain~9%Santarius, Lancet 2009
Burr hole with drain, no irrigation~18%FINISH, Lancet 2024
Burr hole with drain + irrigation~13%FINISH, Lancet 2024
Surgery + MMA embolization~4%EMBOLISE, NEJM 2024
MMA embolization (SQUID) vs. standard16% vs 36% failureSTEM, NEJM 2025

Functional recovery

Roughly 80 percent of patients return to their pre-bleed level of function after surgery, and most of the improvement happens in the first few weeks. Recovery is slower in patients who were already frail, who had severe symptoms on arrival (for example, inability to speak or move one side), or who had large hematomas with significant midline shift.

A short course of inpatient rehab or home physical therapy is common after discharge, especially for patients who were already unsteady on their feet. Driving is usually held for several weeks until the brain has re-expanded and the surgeon clears the patient.

Mortality in the elderly

Short-term surgical mortality is low — typically around 2 to 5 percent. But longer-term survival is more sobering. In population-based studies, the one-year mortality after chronic subdural hematoma in elderly patients is roughly 25 to 30 percent, and at five years it approaches 50 percent. Most of those deaths are not from the hematoma itself but from the underlying conditions that led to the fall in the first place: heart disease, cancer, dementia, and frailty.

What that means in practical terms is that for an older adult, a chronic subdural is a wake-up call, not just a surgical problem. Our team tries to treat it as an opportunity to tune up everything else: review medications, reassess fall risk at home, rebalance blood thinners, and loop in the patient's primary care physician for a broader plan.

The single biggest factor you can control is getting to an experienced team quickly. At UChicago, our trauma and neurovascular surgeons work as one unit — the same on-call group who does a burr hole at 2 a.m. can have an endovascular surgeon ready to embolize the middle meningeal artery the next morning.

References

Santarius T, Kirkpatrick PJ, Ganesan D, et al. Use of drains versus no drains after burr-hole evacuation of chronic subdural haematoma: a randomised controlled trial. Lancet. 2009;374(9695):1067-1073. PMID: 19782872
Kolias AG, Chari A, Santarius T, Hutchinson PJ. Chronic subdural haematoma: modern management and emerging therapies. Nature Reviews Neurology. 2014;10(10):570-578. PMID: 25224156
Almenawer SA, Farrokhyar F, Hong C, et al. Chronic subdural hematoma management: a systematic review and meta-analysis of 34,829 patients. Annals of Surgery. 2014;259(3):449-457. PMID: 24096761
Rughani AI, Lin C, Dumont TM, Penar PL, Horgan MA, Tranmer BI. A case-comparison study of the subdural evacuating port system in treating chronic subdural hematomas. Journal of Neurosurgery. 2010;113(3):609-614. PMID: 20001585
Hutchinson PJ, Edlmann E, Bulters D, et al. Trial of dexamethasone for chronic subdural hematoma. New England Journal of Medicine. 2020;383(27):2616-2627. PMID: 33326713
Raj R, Tommiska P, Koivisto T, et al. Burr-hole drainage with or without irrigation for chronic subdural haematoma (FINISH): a Finnish, nationwide, parallel-group, multicentre, randomised, controlled, non-inferiority trial. Lancet. 2024;403(10446):2798-2806. PMID: 38852600
Davies JM, Knopman J, Mokin M, et al. Adjunctive middle meningeal artery embolization for subdural hematoma. New England Journal of Medicine. 2024;391(20):1890-1900. PMID: 39565988
Liu J, Ni W, Zuo Q, et al. Middle meningeal artery embolization for nonacute subdural hematoma. New England Journal of Medicine. 2024;391(20):1901-1912. PMID: 39565989
Fiorella D, Monteith SJ, Hanel R, et al. Embolization of the middle meningeal artery for chronic subdural hematoma. New England Journal of Medicine. 2025;392(9):855-864. PMID: 39565980

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